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Observing the movement of the baby's sight from two to 24 month of age as they were shown an actress's video invitation to gamble, the investigators found that infants who were later acknowledged to have ASD had more confined visual exposure than their age-mates. Investigators found that the mean height of the eyeballs (i.e. the actor's eyeballs on the screen) was the same in both groups after two month, but after this period the ASD group had decreased it.
Investigators did not suggest that this would be a new test for ASD in infants - a diagnostic of ASD is made by studying a variety of testing and behavioral interaction, usually from the ages of two years (see box). It is encouraging to note that if visual contacts are normally after two month, other features may also be as well.
Thus, it may be possible to avoid the development of AIDS through early action. There is no evidence in this trial to support or refute a parent's claim to having diagnosed AIDS. Significantly, it has provided an expertise that "Autism is a very complicated disease...no two persons with Autism are alike, and therefore a comprehensive diagnostic assessment is needed that considers all facets of a person's behavior".
She also argued that autoimmune disease could be detected in an infant at the ages of two moths, which is not the case. Fact that, unlike what is expected, children with ASD have no impairment of ocular contacting capacity after two moths. It was a coherent trial with neonates aged from two to 24 weeks.
The aim was to find out when the decreased ocular exposure typical of ASD occurred by comparison of the results of 36 month old adolescents with ASD with those with ASD typical of development. Investigators enrolled 59 babies at high risks of ASD (they had a sister child at ASD) and 51 babies at low risks of ASD (no first, second or third grade compared to ASD).
This was to ensure that their group of babies contained some who would be developing ASD. Investigators showed the toddlers a video of an actor they invited to perform and measure the amount of times the toddlers spent looking at their eyeballs, mouths, bodies and bodies using eyetracking devices named orcans.
This test was done 10x at two, three, four, five, six, nine, 12, 15, 18 and 24 month of birth. At 36 month of birth, 13 babies were found to have ASD (12 from the high-risk group and one from the low-risk group). Most of the cases found were masculine.
Investigators likened the eye-tracking results of 11 young men who had been found to have ASD (10 from the high-risk group and one from the low-risk group) to 25 young men who had not (all from the low-risk group). You wanted to see when the decreased ocular exposure, which can be a characteristic of an ASD, was present before apparent signs of an ASD.
In order to avoid distortions of the results, the investigators conducting the test were not informed of which babies were at high or low risks or whether they had already been diagnosed with ASD, and the clinic personnel diagnosing ASD were not informed of the results of the eyes-testing. You found that the mean number of glances was the same in both groups after two month.
In the group that developed, the typical increase in ocular exposure was 3.6% per two to six monthly, while the ASD group showed a decrease in ocular exposure of 4.8% per monthly during this period. Babies who develop usually look more at the sight than at the lips, bodies or things from two to six months, but babies with ASD showed a decrease in ocular focus from two to 24 month, which is half of those who develop babies by 24 year.
The mouth immobilization intensified in the first year and reached its peak in both groups with 18 month. The ASD group showed a less than half reduction in ocular attachment to the human retina compared to typical evolving babies and a 25% increase in stabilization. Likewise, the focus on one property decreased more gradually and rose in the second year.
At 24 month it was twice as high as in typical growing newborns. Early ocular appearance is common in an infant later identified with ASD, but then the values decrease. These contradict earlier assumptions about a lack of adaptability from the moment of delivery - i.e. births of children with ASD are those with "hardwired" bad outcomes.
Instead, the results showed that some socially adaptable behaviors in neonates later detected with ASD may at first be healthy. Development algebra has a high level of so-called three-dimensionality - that is, the "neuronal circuits" can be altered through therapy and workouts. It was a very small trial, suggesting that babies who developed ASD may not have the symptoms of decreased ocular exposure from birth. However, it was not a very small one.
Ocular exposure was recorded in reaction to a female rather than a living person's videotape, and there may have been many other confusing facts that could have explained the results. Diagnosed at 24 month would be preliminary and would be based on a range of signs and not just decreased ocular contacts.
While there is no indication of the effects of a previous diagnostic, this may increase the levels of parent assistance. Scientists suggest that the capacity to fix the eyes at childbirth was abnormal, but decreased by six month.